AIDS and Tuberculosis: A Deadly Liaison by Stefan H. E. Kaufmann, Bruce D. Walker

By Stefan H. E. Kaufmann, Bruce D. Walker

Delivering the most recent details on preventive, diagnostic and healing points of tuberculosis and AIDS, this can be the one e-book to put a tremendous emphasis at the expanding coexistence of those life-threatening ailments in individuals.
Edited by means of impressive scientists within the box, this prepared reference is split into 3 major sections masking immunology and vaccination recommendations, medicinal drugs, and scientific issues.
well timed analyzing for microbiologists, virologists, bacteriologists, immunologists, and pathophysiologists, in addition to for the pharmaceutical and biotechnological industries.

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Extra resources for AIDS and Tuberculosis: A Deadly Liaison

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Infect. , 70, 3592–3601. , Burkhard, M. A. (2005) Pre-existing immunity to pathogenic Listeria monocytogenes does not prevent induction of immune responses to feline immunodeficiency virus by a novel recombinant Listeria monocytogenes vaccine. Vaccine, 23, 1479–1490. , Strobert, E. M. (2007) Live attenuated Listeria monocytogenes expressing HIV Gag: immunogenicity in rhesus monkeys. Vaccine, 25, 7470–7479. , Iijima, N. R. (2008) Novel vaccination protocol with two live mucosal vectors elicits strong cell-mediated immunity in the vagina and protects against vaginal virus challenge.

This might be due to the decreasing amount of antigen provided by the vaccine vectors, as most of these vectors are nonpersistent. Central-memory T-cells, however, are primarily lacking effector functions and require differentiation and expansion in the presence of antigen to respond effectively to virus. However, this might be too slow to prevent the systemic dissemination of HIV. A recent study therefore utilized a SIV protein encoding vector based on rhesus cytomegalovirus (RhCMV); this was chosen because these vectors induce lifelong effector memory T-cell responses.

D. (2006) PD-1 expression on HIV-specific T cells is associated with T-cell exhaustion and disease progression. Nature, 443, 350–354. S. D. (2007) Upregulation of CTLA-4 by HIV-specific CD4 þ T cells correlates with disease progression and defines a reversible immune dysfunction. Nat. , 8, 1246–1254. , Erlich, H. L. (1996) Influence of combinations of human major histocompatibility complex genes on the course of HIV-1 infection. Nat. , 2, 405–411. M. D. (2008) Hla-B57/BÃ 5801 Hiv-1 elite controllers select for rare gag variants associated with reduced viral replication capacity and strong CTL recognition.

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